AbstractPDF
Abstract
Three patients developed hyperglycaemia during tamsulosin use. All patients had diabetes and recovered
after withdrawal. A pharmacological mechanism for this
adverse drug reaction is suggested: stimulation of the
α1-receptor is one of the insulin-independent pathways for glucose uptake, hence inhibition might increase plasma glucose concentrations. Physicians should be aware of hyperglycaemia as possible adverse drug reaction in patients with diabetes using tamsulosin.
